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Sample Publications
Vander Heiden, M.G., & Thompson, C.B. (1999). Bcl-2 proteins: regulators of apoptosis or of mitochondrial homeostasis? Nature Cell Biology, 8, 209-216.
Lindsten, T., Ross, A.J., King, A., Zong, W-X., Rathmell, J.C., Shiels, H.A., Ulrich, E., Waymire, K.G., Mahar, P., Frauwirth, K., Chen, Y., Wei, M., Eng, V.M., Adelman, D.M., Simon, M.C., Ma, A., Golden, J.A., Evan, G., Korsmeyer, S.J., MacGregor, G.R. & Thompson, C.B. (2000). The combined functions of the pro-apoptotic Bcl-2 family members, bak and bax, are essential for normal development of multiple tissues. Molecular Cell, 6, 1389-1399.
Wei, M.C., Zong, W-X., Cheng, E.H.-Y., Lindsten, T., Panoutsakopoulou, V., Ross, A.J., MacGregor, G.R., Thompson, C.B., & Korsmeyer, S.J. (2001). Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death. Science, 292, 727-730.
Zong, W-X., Lindsten, T., Ross, A.J., MacGregor, G.R., & Thompson, C.B. (2001). BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak. Genes & Development, 15, 1481-1486. |
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